Immunobiology, Alopecia Areata

Saturday, 19 June 2010 08:30 - 10:00

Chair Ralf Paus Department of Dermatology, University of Lübeck, Lübeck and School of Translational Medicine, University of ManchesterGermany
Co-Chair Prof Jerry Shapiro Department of Dermatology and Skin Science, University of British ColumbiaCanada
Co-Chair Dr Seiji Arase  Japan
Co-Chair Dr Ulrike Blume-Peytavi  Germany
Significant increase of MAGE3 specific-CTLs in alopecia areata patients
  • Taisuke Ito, Department of Dermatology, Hamamatsu University School of Medicine, Japan
Vitamin D receptor depletion by Wnt/beta-catenin signaling caused alopecia areata in human
  • Beom Joon Kim, Department of Dermatology, Chung-Ang University College of Medicine, Korea
Spreading hair loss in alopecia areata: the domino effect
  • Assoc Prof Stephen Gilmore, Dermatology Research Centre, School of Medicine, University of Queensland., Australia
Early Alopecia Areata is associated with Damage of the Permanent Follicle and Upper Dermis Mast Cell and T Cell Infiltration
  • Prof/Dr Xingqi Zhang, Department of Dermatology, The First Affiliated Hospital, Sun Yat-sen University, China
Unraveling the mysteries of alopecia areata using mouse models
  • Dr John P Sundberg, The Jackson Laboratory, United States
Reduced expression of Vitamin D receptor (VDR) related to alopecia universalis by Wnt/b-catenin signaling pathway
  • Yun Young Lim, Department of Dermatology, Chung-Ang University College of Medicine, Seoul, Korea, Korea
  • Beom Joon Kim, Department of Dermatology, Chung-Ang University College of Medicine, Seoul, Korea, Korea